81. Greenwood Genetic Center - Library Resources Provides explanations of genetics theories and practices using fullmotion videoand Love in Any Language angelman syndrome, 1991, VT QS 675 Lov 1991, 15 min. http://www.ggc.org/library.html

82. Chemistry Of The Cell And Genetics like to try this well written page on cat coat colour genetics angelman syndrome andPraderWilli syndrome are two different conditions both of which seem to be http://www.ucl.ac.uk/~ucbhjow/bmsi/bmsi_4.html

Genetics Lecture 4 Sex determination Surprisingly, it is only in the last 50 years that we have begun to understand the nature of the biological events which determine our sex, (and for that matter, why we bother with sex at all and why two sexes are better than three or more). It is not so long ago that women were blamed if they failed to produce a son for their husband and clearly it was thought that the power of sex determination lay within the body of the woman. During this century the chromosomal basis of human sex determination has been demonstrated and in the last few years some of the genes responsible have been identified. The sexual identity of an individual is determined at several levels, chromosomal sex, gonadal sex, somatic sex and sexual orientation. sex chromosomes The chromosomal basis of sex determination in humans was recognized when metaphase chromosomes from dividing male and female cells could be studied and counted. The normal karyotype contains 46 chromosomes including either two X chromosomes (46XX, females) or one X chromosome and one Y chromosome (46XY, males). Individuals with or karyotypes are female, individuals with

83. To Homepage Of Department To Homepage Of Faculty To Homepage Of of Human genetics 38267275, 1993; Saitoh S, Kubota T, Ohta T, Jinno Y, NiikawaN, Sugimoto T, Wagstaff J, Lalande M Familial angelman syndrome caused by http://www.shinshu-u.ac.jp/souran/menu/gakubu-e/igakub/igakuk/eisei/kubota.html

Takeo Kubota Assistant Professor; M.D.,Ph.D. Department of Hygiene and Medical Genetics Shinshu University School of Medicine E-mail : tkubota@med.shinshu-u.ac.jp Phone : +81-263-37-2618 Fax : +81-263-37-2619 Academic Background School of Medicine, Hokkaido University, 1979-1985 Resident,Department of Pediatrics, School of Medicine, Showa University,1985-90 Assistent Professor, Department of Pediatrics, School of Medicine, Showa University, 1990-91 Research Fellow, Department of Genetics, School of Medicine, Nagasaki University,1991-93 Research Associate, Department of Molecular Genetics, Baylor College of Medicine, 1993-94 Visiting Fellow, National Center for Human Genome Research, NIH, USA, 1994-96 Research Consultant, Center for Medical Genetics, The University of Chicago,1996-97 Assistant Professor, Department of Hygiene and Medical Genetics, School of Medicine, Shinshu University, 1997-present Research and Educational Interests Abnormal DNA methylation causing diseases Genomic Imprinting in congenital diseases Application of Molecular diagnosis for clinics Genetic couselling at genetic clinics Key words: Molecular Genetics, Medical Genetics, DNA methylation, genomic imprinting

84. Emory Genetics Lab, Prader-Willi / Angelman Methylation Studies PraderWilli syndrome (PWS) and angelman syndrome (AS) are clinically distinct, complex EmoryGenetics follows the recommendations of a joint American College http://www.emory.edu/WHSC/GENETICSLAB/dna/prader.htm

PRADER-WILLI / ANGELMAN METHYLATION STUDIES INDICATIONS Click here for Gene Reviews Clinical Summary for AS Click here for Gene Reviews Clinical Summary for PWS Prader-Willi syndrome (PWS) and Angelman syndrome (AS) are clinically distinct, complex disorders, and genetically heterogeneous. Both PWS and AS have been found to result from a deletion in the same area of chromosome 15 (15q11-q13). A deletion in the paternally contributed chromosome will result in PWS, while a deletion in the maternally contributed chromosome will result in AS. For PWS, greater than 70% of cases result from the deletion on chromosome 15 inherited from the father, while 25% occur due to maternal uniparental disomy (UPD). Approximately 5% of PWS cases have an abnormality in the imprinting process (resulting in nonexpression of the paternal genes in the PWS critical region). For AS, greater than 70% of cases result from the deletion on chromosome 15 inherited from the mother, while 3-5% occur due to paternal uniparental disomy (UPD). Another 2-3% of AS patients have an abnormality in the imprinting process. The remaining 25% in this group are hypothesized to have AS on the basis of a single gene mutation whose genetic locus is unknown. In order to determine the likelihood of recurrence for genetic counseling purposes, FISH, and methylation analysis with or without chromosome analysis will be necessary. Emory Genetics follows the recommendations of a joint American College of Medical Genetics/American Society of Human Genetics Test and Technology Transfer Committee, for clinically valid identification and diagnosis of PWS and AS individuals published in 1996*:

85. Kuang-dong Wuu 22 15 16 (1999). SCI. Jiang, Yh, E. Lev-Lehman , J. Bressler, TF Tsai, and ALBeaudet. genetics of angelman syndrome. Am. J. Hum. Genet. 65 1-6 (1999). SCI. http://www.ym.edu.tw/ig/igweb/faculty/tft.html

¡@Institute of Genetics, National Yang Ming University Ting-Fen Tsai, Adjunct Assistant Professor ½²«Fªâ °Æ±Ð±Â Ph.D. Graduate Institute of Microbiology and Immunology, National Yang-Ming University, Taipei, Taiwan Telephone: (02) 2826-7293 Fax: (02)2820-2449 e-mail: tftsai@ym.edu.tw Research Teaching specialities Research specialities: Mouse Genetics Knockout and Transgenic Mice Some recent publications Jiang, Y.-h, T. F. Tsai, J. Bressler, and A. L. Beaudet. Imprinting in Angelman and Prader-Willi syndromes. Curr. Opin. Genet. Dev. 8: 334-342 (1998). SCI Tsai, T. F., M. J. Wu, and T. S. Su. The usage of cryptic splice sites in citrullinemia fibroblast suggests a role of polyadenylation in splice site selection during terminal exon definition. DNACell Biol. 17: 717-725 (1998). SCI Tsai, T. F., A. Raas-Rothschild, Z. Ben-Neriah, and A. L. Beaudet. Prenatal diagnosis and carrier detection for a point mutation in UBE3A causing Angelman syndrome. Am. J. Hum. Genet. 63:1561-1563 (1998). SCI Fang, P., E. Lev-Lehman, T. F. TsaL T. Matsuura, C. S. Benton, J. S. Sutcliff, S. L. Christian, T. Kubota, D. J. Halley, H. Meijers-Heijboer, S. Langlois, J. M. Graham, Jr., J. Beuten, P. J. Willems, D. H. Ledbetter, and A. L. Beaudet. The spectrum of mutations in UBE3A causing Angelman syndrome. Hum. Mol. Genet. 8: 129-135 (1999). SCI

86. PEDS: Division Of Genetics: Fluorescent In Situ Hybridization (FISH) genetics Menu Choose a genetics Resource. angelman syndrome D15S10 locus http://peds.wustl.edu/div/genetics/spec/cyto/mc_fish.html

Department of Pediatrics or view Menu: Choose a Genetics Resource General Information Clinical Activities Information for Parents Faculty Summaries Bookmark this site Contact us Billing questions Accreditation Download the Cytogenetics and Molecular Cytogenetics requisition form Fluorescent In Situ Hybridization (FISH) FISH may be performed on either chromosomes or interphase nuclei in cultured or uncultured cells from amniotic fluid, chorionic villi, bone marrow, peripheral blood and fibroblasts. Full information regarding the FISH assay requested must be indicated on the requisition form. Please call the Cytogenetics Lab at (314) 454-8101 for any questions about FISH. List of Available Tests Angelman syndrome [D15S10 locus] Cultured peripheral blood lymphocytes are processed as for a routine cytogenetic study and analyzed by the investigative method FISH. The D15S10 cosmid (Vysis, Inc.) and a control probe, D15Z1 (Vysis, Inc.) which tag chromosomes 15 are hybridized to metaphase chromosomes. The D15S10 probe has been found deleted in a high proportion of patients with Prader Willi syndrome, Angelman syndrome or in other disorders with similar phenotypes. Although the probe is from the smallest region of overlap of the deletions in these patients, it is not known to be the specific gene which causes the phenotype.

87. Angelman Syndrome - Description, Links And Books angelman syndrome, description, links and books Clinical Features of. angelman syndrome EM, Zori RT. angelman syndrome Consensus for diagnostic criteria. and Handbooks. angelman syndrome. angelman syndrome, a parent's http://www.isn.net/~jypsy/angelman.htm

Clinical Features of Angelman Syndrome Consistent (100%) Developmental delay, functionally severe Speech impairment, none or minimal use of words; receptive and non-verbal communication skills higher than verbal ones Movement or balance disorder, usually ataxia of gait and/or tremulous movement of limbs Behavioral uniqueness: any combination of frequent laughter/smiling; apparent happy demeanor; easily excitable personality, often with hand flapping movements; hypermotoric behavior; short attention span Delayed, disproportionate growth in head circumference, usually resulting in Microcephaly (absolute or relative) by age 2 Abnormal EEC, characteristic pattern with large amplitude slow-spike waves Associated (20 - 80%) Strabismus Hypopigmented skin and eyes Tongue thrusting; suck/swallowing disorders Hyperactive tendon reflexes Feeding problems during infancy Uplifted, flexed arms during walking Prominent mandible Increased sensitivity to heat Wide mouth, wide-spaced teeth

88. Genzyme Corporation - Genetics Li, L, et al. Carrier frequency of the Bloom syndrome blmAsh mutation in the AshkenaziJewish population. Molecular genetics Metabolism 6428690(1998). http://www.genzymegenetics.com/genetics/clinicalinfo/molgen/bloom.htm

89. Marfan Syndrome Marfan syndrome up. Marfan syndrome / genetics Marfan syndrome /genetics. GeneReviews congenital contractural arachnodactyly, http://omni.ac.uk/browse/mesh/detail/C0024796L0024796.html

Marfan Syndrome [up] Marfan Syndrome / genetics Related topics: broader Abnormalities, Multiple Connective Tissue Diseases Genetic Diseases, Inborn Heart Defects, Congenital other Angelman Syndrome Ataxia Telangiectasia Cellulitis Collagen Diseases ... Marfan Association UK The Web site for the Marfan Association in the UK, providing a fact sheet for patients about the syndrome, news, events, a contact list of support groups, and a list of publications. Charities Great Britain Marfan Syndrome Marfan Syndrome / genetics GeneReviews : congenital contractural arachnodactyly Notes for physicians on congenital contractural arachnodactyly (CCA, Beals Syndrome). This document includes diagnosis, a clinical description, differential diagnosis, management, genetic counselling, and molecular genetics. Posted in January 2001, this resource forms part of GeneReviews (formerly GeneClinics profile), a peer-reviewed clinical genetic information resource that is funded by the US National Institutes of Health (NIH) and produced by the University of Washington, Seattle. This resource contains a summary and bibliographical references of the review. Free access to the full-text version of the review requires brief registration. Marfan Syndrome / genetics Last modified 28/Mar/2003 [Low Graphics]

90. Uniparental Disomy: Prader-Willi Syndrome, Angelman Syndrome - Lucile Packard Ch Medical genetics. Uniparental Disomy PraderWilli syndrome, Angelmansyndrome What is uniparental disomy? Normally, we inherit one http://www.lpch.org/DiseaseHealthInfo/HealthLibrary/genetics/uniparen.html

Genetics Clinic Metabolic/Biochemical Genetics Clinic Medical Geneticists Medical Genetics Medical Genetics Uniparental Disomy: Prader-Willi Syndrome, Angelman Syndrome What is uniparental disomy? Normally, we inherit one copy of each chromosome pair from our biological mother, and the other copy of the chromosome pair from our biological father. Uniparental disomy refers to the situation in which two copies of a chromosome come from the same parent, instead of one copy coming from the mother and one copy coming from the father. Angelman syndrome and Prader-Willi syndrome are examples of disorders caused by uniparental disomy. What is Angelman syndrome (AS)? People with Angelman syndrome (AS) have mental retardation, severe speech problems, stiff arm movements, and a stiff, uncoordinated walk. They may have seizures and often have inappropriate outbursts of laughter. Angelman syndrome results when a baby inherits both copies of chromosome #15 from the father (rather than one from the mother and one from the father). What is Prader-Willi syndrome (PWS)?

91. Angelman Syndrome Lalande M. Parental imprinting and human disease. Annual Review of Genetics1997;30173195. McKusick VA. Happy Puppet syndrome. angelman syndrome. http://ibis-birthdefects.org/start/ukrainian/uangel.htm

Please set browser to encoding in cyrillic) ("Ðàä³ñíî¿ ìàð³îíåòêè" ñèíäðîì) "В новинах" Ðîçóìîâà â³äñòàë³ñòü, çàãàëüíèé ìîâíèé íåäîðîçâèòîê âàæêîãî ñòóïåíþ, õàðàêòåðíà àòàêòè÷íà õîäà ("õîäà ìåõàí³÷íî¿ ëÿëüêè": êðîêóâàííÿ ³ç ç³ãíóòèìè â ë³êòÿõ ðóêàìè), ì³êðîöåôàë³ÿ, ñïëîùåíà ïîòèëèöÿ, íèæí³é (ìàíäèáóëÿðíèé) ïðîãíàòèçì, ïðîòðóç³ÿ ÿçèêà, ïàðîêñèçìè íåìîòèâîâàíîãî ñì³õó, äèôóçí³ çì³íè íà ÅÅ. Ñèíäðîì Àíãåëüìàíà â³äíîñèòüñÿ ëî õâîðîá, îáóìîâëåíèõ ãåíîìíèì ³ìïðèíòèíãîì (â³ä àíãë. "imprint" - "â³äáèòîê"). Ïðè÷èíîþ ìîæå áóòè óñïàäêîâàíà â³ä ìàòåð³ äåëåö³ÿ 15-¿ õðîìîñîìè (q11-q13), áàòüê³âñüêà äèñîì³ÿ 15 (â 3%-5% âèïàäê³â), òî÷êîâà ìóòàö³ÿ àêòèâíîãî àëåëÿ àáî ìóòàö³ÿ ³ìïðèíòèíãîâîãî öåíòðó. ANCR (Angelman Syndrome Chromosome Region) êàðòîâàíèé íà 15(q11-q13). Ðàííº íàâ÷àííÿ "ìîâ³ æåñò³â" òà òðåíóâàííÿ íåâåðáàëüíèõ íàâèê³â ìîæå äåùî ïîêðàùèòè ïåðñïåêòèâó òàêèõ ä³òåé â ïëàí³ ñîö³àëüíî¿ àäàïòàö³¿. Ïðîãíîç ùîäî ðîçâèòêó ìîâè - íåñïðèÿòëèâèé. Á³ëüø³ñòü õâîðèõ íå ìàþòü íàâèê³â ñàìîîáñëóãîâóâàííÿ ³ ïîòðåáóþòü ñòîðîííüî¿ äîïîìîãè ïðîòÿãîì ö³ëîãî æèòòÿ. Greenberg F. Contiguous gene syndrome. Growth: Genetics and Hormones 1993;9:5-10.

92. Medical Genetics - Uniparental Disomy: Prader-Willi Syndrome, Angelman Syndrome Medical genetics Uniparental Disomy PraderWilli syndrome, Angelmansyndrome. What is uniparental disomy? Normally, we inherit one http://www.mmhs.com/clinical/peds/english/genetics/uniparen.htm

English - Adult English - Pediatric Spanish - Adult Spanish - Pediatric Medical Genetics Uniparental Disomy: Prader-Willi Syndrome, Angelman Syndrome What is uniparental disomy? Normally, we inherit one copy of each chromosome pair from our biological mother, and the other copy of the chromosome pair from our biological father. Uniparental disomy refers to the situation in which two copies of a chromosome come from the same parent, instead of one copy coming from the mother and one copy coming from the father. Angelman syndrome and Prader-Willi syndrome are examples of disorders caused by uniparental disomy. What is Angelman syndrome (AS)? People with Angelman syndrome (AS) have mental retardation, severe speech problems, stiff arm movements, and a stiff, uncoordinated walk. They may have seizures and often have inappropriate outbursts of laughter. Angelman syndrome results when a baby inherits both copies of chromosome #15 from the father (rather than one from the mother and one from the father). What is Prader-Willi syndrome (PWS)?

93. Angelmanin Oireyhtymä - WWW-sivuja, Kirjallisuutta Laan, LA den Boer A, Hennekam R., Renier W. Brouwer 0. (1996) angelman Syndromein Adulthood. American Journal of Medical genetics, 66, 356360. http://www.kvtl.fi/angel/linkit.htm

L http://chem-faculty.ucsd.edu/harvey/asfsite/Diagnostic_Criteria.html http://www.faseb.org/genetics/acmg/pol-22.htm http://www3.ncbi.nlm.nih.gov/htbin-post/Omim/dispmim?105830 Online Mendelian Inheritance in Man-tietokannan artikkeli. http://www.ncbi.nlm.nih.gov/entrez/query.fcgi http://www.mygale.org/02/angelman/index-2.htm Bibliografia. http://chem-faculty.ucsd.edu/harvey/angelpro.html http://www.geneclinics.org/profiles/angelman P http://www.asclepius.com/iaso http://www.asclepius.com/angel/ http://chem-faculty.ucsd.edu/harvey/AS_info.html http://people.zeelandnet.nl/fhof/angelman.htm Hollantilaisen Frank van Hofin sivuilla on mm. Angelman-lasten valokuvagalleria ja amerikkalaisen Martha Sprowlesin kirjoittama viesti uusille Angelman-perheille. http://chem-faculty.ucsd.edu/harvey/asfsite http://www.australianholidays.com/asa/asahome.htm Australian Angelman-yhdistyksen sivut. http://shell.idt.net/~julhyman/angel.htm Amerikkalainen Julie Hyman on koonnut mm. runoja ja niksikirjan "Angelman Syndrome From A to Z". http://www.multimania.com/angelman (uusi osoite 1.11.1999)

94. Angelman Syndrome; Treatment, Prevention, Cure angelman syndrome Search here for information which may include treatment, diagnosis,prevention, support groups, email lists, messageboards, personal stories http://www.healthlinkusa.com/content/18.html

Latest Health News Find Drug Information Health Calculators All Words Any Words A B C D ... Z Sunday, March 30, 2003 Alternative therapy struggles to bridge East-West divide Click here to read more Doctors are not following guidelines recommending flu and pneumonia vaccinations for hospitalized adults, leaving millions of elderly patients vulnerable to potentially deadly ailment Click here to read more A helping hand for uninsured; Agencies find ways to provide health care for area children Click here to read more The government has approved the first generic version of the acne drug, Accutane Click here to read more Gates to give India $100m for AIDS; Microsoft CEO begins controversy-laced trip to fight virus Click here to read more Allergies and Asthma Alternative Medicine Arthritis and Your Health ... Women's Health Thursday April 03, 2003 Spina Bifida: Spina Bifida is a rare birth disorder, affecting approximately 1 in 12-1400 live births. Click here to learn more Craniosynostosis: Sagittal Synostosis...males are affected about three times as often as females. Click here to learn more Spinal Cord Injury: Acts of violence have now overtaken falls as the second most common source of spinal cord injury.

95. Daniel J. Driscoll, M.D., Ph.D., Faculty, Molecular Genetics And Microbiology, U Prenatal Diagnosis 20300306, 2000. Lossie, AC and Driscoll, DJ Transmission ofAngelman syndrome by an affected mother.genetics in Medicine 1262-266, 1999. http://www.mgm.ufl.edu/faculty/Ddriscoll.htm

Your browser does not support script Daniel J. Driscoll, Professor M.D Albany Medical College Ph.D. Indiana University School of Medicine M.S. Rutgers University A.B. Colgate University Postdoctoral Genetics Fellow, Johns Hopkins University Pediatric Residency, Johns Hopkins University Awards and Professional Services NIH Predoctoral Trainee, Indiana University School of Medicine The Richard T. Beebe Award in Medicine, Albany Medical College NIH Postdoctoral Fellowship, The Johns Hopkins Hospital Finalist, Postdoctoral Basic Research Award, American Society of Human Genetics Basil O'Connor Starter Scholar Research Award, March of Dimes The John T. and Winifred M. Hayward Professorship in Genetics Research, University of Florida

96. Prader-Willi Syndrome Kubota T, et al. Methylationspecific PCR simplifies imprinting analysis. NatureGenetics 1997; 1616-17. References for angelman syndrome. • GeneClinic. http://www.aruplab.com/testbltn/angelman.htm

For technical information, contact: Jamie McDonald, M.S. Genetic Counselor, DNA Diagnostics Laboratory, University of Utah For scientific and clinical information, contact: Rong Mao, M.D. Assistant Medical Director, DNA Diagnostics Laboratory, University of Utah Kenneth Ward, M.D. Medical Director, DNA Diagnostics Laboratory, University of Utah References for Prader-Willi Syndrome • GeneClinic. http://www.geneclinic.org/ Prader-Willi Syndrome. • Kosaki K, et al. Prader-Willi and Angelman syndrome: diagnosis with a bisulfite-treated methylation specific PCR method. Am J Med Genet 1997; 73(3):308-313. • Kubota T, et al. Methylation-specific PCR simplifies imprinting analysis. Nature Genetics 1997; 16:16-17. References for Angelman Syndrome • GeneClinic. http://www.geneclinic.org/ Angelman Syndrome. • Kosaki K, et al. Prader-Willi and Angelman syndrome: diagnosis with a bisulfite-treated methylation specific PCR method. Am J Med Genet 1997; 73(3):308-313. • Kubota T, et al. Methylation-specific PCR simplifies imprinting analysis. Nature Genetics 1997; 16:16-17. ARUP Laboratories 500 Chipeta Way Salt Lake City, UT 84108

97. Angelman Syndrome Methylation angelman syndrome Methylation. DESCRIPTION angelman Xbal/Notl. Presenceof only the paternal band supports a diagnosis of angelman syndrome. http://www.shodairhospital.org/angelman syndrome.htm

Contact Us Foundation Genetics Home ... Staff Angelman Syndrome Methylation DESCRIPTION: Angelman syndrome is a disorder which manifests with severe developmental delays, absence of speech, ataxia, seizures, and inappropriate bouts of laughter. The disorder is caused by lack of maternal expression of genes in the q1 I segment of chromosome 15. More than 70% of patients lack a maternal-specific pattern of methylation (imprint) at chromosome 15q I I -q 13 due to deletion, paternal uniparental disomy, or imprinting mutation. Remaining patients may have mutation of the UBE3A gene (Matsuura et al., Nat Gen 1997, 15:74-77), however clinical testing is not currently available. REASONS FOR REFERRAL Methylation testing can confirm a diagnosis of Angelman syndrome in a chiJd with consistent symptoms and signs as described above. According to "Diagnostic Testing for Prader-Willi and Angelman Syndromes" (Am J Hum Genet "If biparental inheritance is identified (by methylation)...most identifiable cases of Angelman syndrome are…ruled out."

98. Show_part the imprinted human PraderWilli syndrome regionGenes Dev9 contig of the Prader-Willi/Angelmanchromosome region Research into the genetics of autism and other http://medschool.mc.vanderbilt.edu/cmn/php_files/show_partcmn.php?id3=880

99. Birth Defect Syndromes Birth Defects genetics Center. Diagnosis Syndromes - Issues http://www.southalabama.edu/genetics/gdrophealth.htm

Birth Defects Genetics Center Diagnosis - Syndromes - Issues: Angelman Syndrome CP Cerebral Palsy Down Syndrome ... More This site offers information mostly for educational purposes. This site is not intended to alter health care protocols nor to serve as a sole source of medical information. Please read Always seek the advice of your local health care provider. Questions? Home Vistor # Department of Medical Genetics University of South Alabama Email: Webmaster Sponsor: University of South Alabama Site Concept: W. W. Page Concept: W. W. 2000720 R.C.

100. Medical Genetics - Non-Traditional Inheritance If you cannot find the information in which you are interested, please visit theMedical genetics Online Resources page in this Web site for an Internet/World http://www.mccg.org/childrenshealth/genetics/nontrad.asp

About MCCG News MCCG Careers Health Careers ... Home You are here Home Children's Health Medical Genetics Inside Children's Health SEARCH Children's Health Children's Health Home Adolescent Medicine Allergy, Asthma and Immunology Pediatric Arthritis and Other Rheumatic Diseases Burns Cardiovascular Disorders Craniofacial Anomalies Dental and Oral Health Dermatology Diabetes and Other Endocrine Metabolic Disorders Digestive and Liver Disorders Ear, Nose and Throat Eye Care Medical Genetics Growth and Development Hematology and Blood Disorders High-Risk Newborn High-Risk Pregnancy Infectious Diseases Child and Adolescent Mental Health Neurological Disorders Normal Newborn Oncology Orthopaedics Common Childhood Injuries and Poisonings Pregnancy and Childbirth Respiratory Disorders Safety and Injury Prevention The Child Having Surgery Care of the Terminally Ill Child Transplantation Genitourinary and Kidney Disorders Non-Traditional Inheritance Inheritance patterns which do not fall into the chromosomal, single gene, or multifactorial categories, are referred to as “non-traditional”. Listed in the directory below are some examples, for which we have provided a brief overview. If you cannot find the information in which you are interested, please visit the

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