21. A Cluster Of Creutzfeldt-Jacob Disease Patients From Nassau A Cluster of creutzfeldtjacob disease Patients from Nassau County, New York,USA. February, 2001 Annals of Clinical and Laboratory Science Vol. 31 no. http://www.organicconsumers.org/madcow/cluster501.cfm

News Campaigns GE Food Organics ... email this page A Cluster of Creutzfeldt-Jacob Disease Patients from Nassau County, New York, USA February, 2001 Annals of Clinical and Laboratory Science Vol. 31 no. 2 p. 211-2 Creutzfeldt-Jacob Disease (CJD) is a rare and transmissible neurological disorder, which has been increasing in frequency in the United States over the past two decades. [...] We report 7 cases of CJD from North Shore University Hospital, a community-based teaching institution that serves Nassau County on Long Island, NY. These cases were diagnosed and died during the 12-mo period between mid-June 1999 to midJune 2000... These data suggest a CJD death rate in Nassau County... approximately 6 times the national rate. During the previous 1-yr period, no case of CJD was diagnosed in our laboratory [...] The authors are concerned that the number of CJD cases in our catchment area appears to have increased dramatically during the 12-mo period. [...] News Campaigns GE Food Organics ... Site Map Organic Consumers Association 6101 Cliff Estate Rd, Little Marais, MN 55614

22. Creutzfeldt-Jacob Disease And Other Spongiform Encephalopathies - New Treatments spongiform encephalopathy, and kuru); and others have been transmitted by inheritanceof mutations in creutzfeldtjacob disease, spongiform encephalopathies http://www.medical-library.org/journals2a/creutzfeldt_jacob2.htm

This page has moved. Click here to view. Creutzfeldt-Jakob Disease and Related Transmissible Spongiform Encephalopathies Creutzfeldt-Jakob disease is the major transmissible spongiform encephalopathy (or prion disease) in humans. The incidence of Creutzfeldt-Jakob disease has not changed in Transmissible Spongiform Encephalopathies Scrapie is a subacute, progressive ataxia of sheep and goats. Animals affected by scrapie have been recognized by shepherds for over 200 years, and for many years the disorder was regarded as an inherited, degenerative disease of the brain and spinal cord. In 1936 scrapie was reported to be transmitted from sheep to sheep with an incubation period in excess of one year; nevertheless, controversy continues to this day about the natural mode of transmission and the relative role of genetic susceptibility. The disease has been experimentally transmitted to many species, but although amplification occurs in some neural cell lines, no in vitro assay has been developed. Therefore, most of our knowledge of the nature of prions comes from Recurrent outbreaks of transmissible spongiform encephalopathy have occurred on mink ranches in Wisconsin, and isolated outbreaks have been reported in Canada, Finland, Germany, and Russia. Feed contaminated with tissue from scrapie-affected sheep was assumed to be the mode of transmission, but recent observations suggest that tissue from other infected animal species may also have been involved. Chronic wasting disease of deer and elk is a transmissible spongiform encephalopathy found in captive animals in the western United States and

23. ISG Website:  Image Processing For The Detection Of Creutzfeldt-Jacob Disease: Image processing for the detection of creutzfeldtjacob disease / Home / research/ image processing for the detection of creutzfeldt-jacob disease /. http://www.ee.ed.ac.uk/~neural/oldISG/research/cjd.html

Home Research Conferences Publications ... ISG Talks Image processing for the detection of Creutzfeldt-Jacob disease: Home research / image processing for the detection of creutzfeldt-jacob disease / CJD Diagnosis using MRI Diagnosis of CJD Creutzfeld-Jakob disease (CJD) is a rare fatal transmissible illness that presents, in most cases, with rapidly evolving dementia including ataxia, myoclonia and altered higher cortical function. Due to symptomatic similarity with other neurological disorders it can be a difficult condition to diagnose and diagnostic confirmation can only currently be made post-mortem via neuropathological examination. Early evidence suggested Magnetic Resonance Imaging (MRI) may provide a route to in-vivo diagnosis of CJD and more promising results have since been observed in the MRI data for patients suffering from new-variant CJD (nvCJD) which is the human prion disease suspected to be causally linked to Bovine Spongiform Encephalopathy (BSE) in cattle. Through the analysis of a large set of post-mortem MR images of the human brain, collected from patients that presented with symptoms of neurological disorder, the aim of this study is to investigate the potential of MRI data for use as an indicator of condition in suspected cases of CJD or nvCJD. Project participants: Mark Harding Website last rebuilt, by

24. Iatrogenic Creutzfeldt-Jacob Disease - General Practice Notebook medical information from General Practice Notebook. iatrogenic CreutzfeldtJacobdisease. The use of prion-contaminated medication http://www.gpnotebook.co.uk/cache/1067057225.htm

iatrogenic Creutzfeldt-Jacob disease The use of prion-contaminated medication, grafts and instruements may result in iatrogenic Creutzfeldt-Jacob disease (CJD). The clinical features of the disease vary according to the route of innoculation. Central innoculation, such as an infected dural graft, results in a rapidly progressive neurodegenerative disease similar to sporadic CJD, characterised by: ataxia dementia myoclonus rididity akinetic mutism survival is 2-12 months from presentation Peripheral innoculation, such as the old human growth hormone products, results in a slightly less acute disease disease characterised by: progressive cerebellar ataxia dementia is not invariably present survival is 8-18 months from presentation Click here for more information...

25. Creutzfeldt-Jacob Disease (iatrogenic) - General Practice Notebook medical information from General Practice Notebook. CreutzfeldtJacobdisease (iatrogenic). The use of prion-contaminated medication http://www.gpnotebook.co.uk/cache/999948361.htm

Creutzfeldt-Jacob disease (iatrogenic) The use of prion-contaminated medication, grafts and instruements may result in iatrogenic Creutzfeldt-Jacob disease (CJD). The clinical features of the disease vary according to the route of innoculation. Central innoculation, such as an infected dural graft, results in a rapidly progressive neurodegenerative disease similar to sporadic CJD, characterised by: ataxia dementia myoclonus rididity akinetic mutism survival is 2-12 months from presentation Peripheral innoculation, such as the old human growth hormone products, results in a slightly less acute disease disease characterised by: progressive cerebellar ataxia dementia is not invariably present survival is 8-18 months from presentation Click here for more information...

26. Creutzfeldt-Jacob Disease I. First Previous Next Last Index Home Text, Slide 15 of 30. http://www.biomedicine.dote.hu/cselenyi/phd3/sld015.htm

27. Creutzfeldt-Jacob Disease II. First Previous Next Last Index Home Text, Slide 16 of 30. http://www.biomedicine.dote.hu/cselenyi/phd3/sld016.htm

28. Brain - Creutzfeldt-jacob Disease (cjd) (male) creutzfeldtjacob disease (CJD) creutzfeldt-jacob disease (CJD) isone of a group of diseases called spongiform encephalopathies. http://www.thehealthyforum.com/bodymapssec/brain_cjd.html

the healthyforum .com The information available on this web site is not a substitute for diagnosis and advice from your qualified practitioner. You must always consult an appropriate specialist in order to address your personal requirements. Please see our Terms and conditions of use and Privacy statement. print this page email to a friend Creutzfeldt-Jacob Disease (CJD) Creutzfeldt-Jacob Disease (CJD) is one of a group of diseases called spongiform encephalopathies. (Spongiform because the affected brain resembles a sponge, with lots of cystic spaces, while encephalopathy simply means a problem causing structural change in the brain). These include BSE in cattle and Scrapie in sheep. There is rapidly progressive degeneration of brain tissue. In the human this causes dementia and jerking of the limbs. The cause is thought to be an abnormal protein called a prion. Our cells contain a protein called PrP. Prions are made as a result of a mutation in the gene that codes for this protein. They are thought to be able to reproduce themselves and are incorporated into cell walls, where they cause the cell to degenerate. return to mood disorders return to body map home WIS ... contact us

29. Brain - Creutzfeldt-jacob Disease (cjd) (female) creutzfeldtjacob disease (CJD) Creutzfeldt-Jacobs Disease (CJD) isone of a group of diseases called spongiform encephalopathies. http://www.thehealthyforum.com/bodymapssec/brain_cjdf.html

the healthyforum .com The information available on this web site is not a substitute for diagnosis and advice from your qualified practitioner. You must always consult an appropriate specialist in order to address your personal requirements. Please see our Terms and conditions of use and Privacy statement. print this page email to a friend Creutzfeldt-Jacob Disease (CJD) Creutzfeldt-Jacobs Disease (CJD) is one of a group of diseases called spongiform encephalopathies. (Spongiform because the affected brain resembles a sponge, with lots of cystic spaces, while encephalopathy simply means a problem causing structural change in the brain). These include BSE in cattle and Scrapie in sheep. There is rapidly progressive degeneration of brain tissue. In the human this causes dementia and jerking of the limbs. The cause is thought to be an abnormal protein called a prion. Our cells contain a protein called PrP. Prions are made as a result of a mutation in the gene that codes for this protein. They are thought to be able to reproduce themselves and are incorporated into cell walls, where they cause the cell to degenerate. return to mood disorders return to body map home WIS ... contact us

30. Creutzfeldt-Jacob Disease - CJD What Is CreutzfeldtJakob Disease? Who Can Be Stricken By Creutzfeldt-JakobDisease? Creutzfeldt-Jakob Disease can afflict anyone. http://thailabonline.com/brain1cjd.htm

BECOME A MEMBER Shopping Health Products - à¤×èͧÇÑ´¤ÇÒÁà¢éÁáʧ Photometer - - ¡Åéͧ¨Øŷȹì - Microscope - - à¤×èͧ´Ù´»ÔÁÒ³ÊÒ/Autopipette - - ªØ´ªèÇÂà¨ÒÐàÅ×Í´»ÅÒ¹ÔéÇ /Lancet - - ªØ´µÇ¨¡ÒµÑ駤Àì/ Pregnancy - - ªØ´µÇ¨ËÒÊÒàʾµÔ´-DrugAbuse - - ªØ´µÇ¨äÇÑʵѺÍÑ¡àʺ/Hepatitis B - - ªØ´µÇ¨äÇÑʵѺÍÑ¡àʺ/Hepatitis C - - ªØ´µÇ¨ËÒÇѹä¢èµ¡/ Ovulation test - - ªØ´µÇ¨¡Í§¡Òà¡Ô´ÁÐàç§/ Tumor- - ªØ´µÇ¨ËÒ¡ÒµÔ´àª×éÍäÇÑÊ HIV - - ªØ´µÇ¨¡ÒµÔ´àª×éÍ«Ô¿ÔÅÔÊ /VDRL - - ªØ´µÇ¨¡ÒµÔ´àª×éÍÁÒÅÒàÕÂ/ Malaria - - ¹éÓÂҵǨËÒËÁÙèâÅËÔµ ABO / Rh - - ªØ´·´Êͺâ´ÂÇÔ¸Õ EIA - top Creutzfeldt-Jakob Dissease (CJD) VARIANT CREUTZFELDT-JAKOB DISEASE (vCJD) Health Navigation Contact : info@thailabonline.com

31. UPMC Health System News Bureau News Bureau News Bureau Home Page. creutzfeldtjacob disease. INFORMATIONAL RELEASE. http://www.upmc.edu/NewsBureau/director/cjd_release.htm

News Bureau News Bureau Home Page In The News News Releases Archives Biographies Contact the News Bureau CREUTZFELDT-JACOB DISEASE INFORMATIONAL RELEASE PITTSBURGH, June 12, 2002 - In the interest of public disclosure, UPMC Health System is releasing the following information about about a former surgical patient who, upon autopsy, was determined to have Creutzfeldt-Jacob Disease ( CJD In April 2001 a patient had surgery at UPMC Presbyterian to treat a condition of the nervous system. The surgical instruments were sterilized with standard procedures following the surgery. In the first quarter of 2002, the patient died from progression of a neurological disorder. Following the patient's death it was determined upon autopsy, for the first time, that the patient had Creutzfeldt-Jacob Disease (CJD). This disease is believed to be transmitted by an infectious protein, or prion. Prions may not be eliminated by standard instrument sterilization processes. The instruments used in the patient's surgery presented an extremely low risk of transmitting CJD to patients who had neurosurgery with these instruments during the period from April 17, 2001 to April 6, 2002. There are only a very small number of reported cases of CJD developing after exposure of a patient to instruments used in a prior surgery on an individual with CJD, the most recent being in Europe in 1976. No such cases have been reported in the United States since the adoption of the standard sterilization techniques which were in use at the time of the patient's operation in April of 2001.

32. Creutzfeldt-Jacob Disease creutzfeldtjacob disease. Definition creutzfeldt-jacob disease is adisorder involving rapid decrease of mental function and movement. http://www.umm.edu/ency/article/000788.htm

Home Medical Reference Encyclopedia (English) Toggle English Spanish Creutzfeldt-Jacob disease Overview Symptoms Treatment Prevention Definition: Creutzfeldt-Jacob disease is a disorder involving rapid decrease of mental function and movement. These are abnormalities believed to be caused by damage done to the brain by a protein called a prion. This protein folds abnormally, and seems to encourage other proteins to become similarly misshapen, affecting their ability to function. Alternative Names: Transmissible Spongiform Encephalopathy; New variant CJD - "the human form of mad cow disease" Causes, incidence, and risk factors: Creutzfeldt-Jakob disease is a disease which can occur sporadically (for no known reason), as a result of exposure to contaminated products. Rarely, it can be genetically inherited. The disorder itself is rare, occurring in about 1 out of 1,000,000 people. It usually first appears in midlife, beginning between ages 20 and 70, with average age at onset of symptoms being in the late 50's. However, some cases have occurred in adolescents who have received

33. The Dictionary Of Cell And Molecular Biology - Online! Result of search for 1578. Welcome, new user. creutzfeldtjacob disease. Rarefatal presenile dementia of humans, similar to kuru and other slow viruses. http://www.mblab.gla.ac.uk/~julian/dict2.cgi?1578

34. Whitetails Unlimited Is Leading The Fight Against Chronic Wasting Disease bovine spongiform encephalopathy (known as mad cow disease); scrapie, which hasaffected sheep for some 200 years; and creutzfeldtjacob disease in humans. http://www.whitetailsunlimited.com/pages/cwd/HumanHealth.htm

OFFICIAL WEBSITE OF WHITETAIL ENTHUSIASTS View Cart Privacy Policy A NATIONAL ORGANIZATION WORKING FOR AN AMERICAN TRADITION The Human Health Question The World Health Organization and the federal Centers for Disease Control agree that there is no scientific evidence CWD can infect humans. CWD is in a class of diseases called transmissible spongiform encephalopathies. In addition to CWD, there are three other variations that affect animals: bovine spongiform encephalopathy (known as mad cow disease); scrapie, which has affected sheep for some 200 years; and Creutzfeldt-Jacob disease in humans. There are two variations of the disease in humans. Creutzfeldt-Jacob disease occurs naturally from unknown causes in about one out of every million people. That rate has remained constant over time and geography, and is not connected to CWD. Variant Creutzfeldt-Jacob disease, which has been linked to the large-scale outbreak of Mad Cow Disease in cattle herds in Great Britain, is identifiably different from the classic CJD. During the Mad Cow outbreak in England, the British population consumed an estimated 750,000 infected cattle during a 10-year period. Only 100 humans have contracted Variant Creutzfeldt-Jacob disease to date. However, WHO also says no part of a deer or elk with evidence of CWD should be eaten by people or other animals. (It is common sense that you should not consume any animal known to be diseased.) Over 16 years of monitoring Colorado's infected area has resulted in no evidence that CWD has transferred to people or cattle living there.

35. Health Library Find Information On Creutzfeldt-Jacob Disease At creutzfeldtjacob disease. Definition creutzfeldt-jacob disease is adisorder involving rapid decrease of mental function and movement. http://www.mercksource.com/pp/us/cns/cns_hl_adam.jspzQzpgzEzzSzppdocszSzuszSzcns

36. Welcome To ENH.org - Health Encyclopedia: Creutzfeldt-Jacob Disease creutzfeldtjacob disease. Definition creutzfeldt-jacob disease is adisorder involving rapid decrease of mental function and movement. http://www.enh.org/Encyclopedia/ency/article/000788.asp

Disease Reference Injury Reference Test Reference Nutrition Reference ... Symptoms Reference Creutzfeldt-Jacob disease Disease Injury Nutrition Poison ... Z Definition: Creutzfeldt-Jacob disease is a disorder involving rapid decrease of mental function and movement. These are abnormalities believed to be caused by damage done to the brain by a protein called a prion. This protein folds abnormally, and seems to encourage other proteins to become similarly misshapen, affecting their ability to function. Alternative Names: Transmissible Spongiform Encephalopathy; New variant CJD - "the human form of mad cow disease" Causes, incidence, and risk factors: Creutzfeldt-Jakob disease is a disease which can occur sporadically (for no known reason), as a result of exposure to contaminated products. Rarely, it can be genetically inherited. The disorder itself is rare, occurring in about 1 out of 1,000,000 people. It usually first appears in midlife, beginning between ages 20 and 70, with average age at onset of symptoms being in the late 50's. However, some cases have occurred in adolescents who have received

37. EID V3 N1: The Epidemiology Of Creutzfeldt-Jakob Disease In Canada: A Review Of 1), which uses mortality data and active surveillance methods, has provided furtherinformation on the epidemiology of creutzfeldtjacob disease (CJD). http://www.cdc.gov/ncidod/eid/vol3no1/stratton.htm

Dispatches The Epidemiology of Creutzfeldt-Jakob Disease in Canada: A Review of Mortality Data Download Article Creutzfeldt-Jakob disease (CJD), and particularly its transmissibility through blood and blood products, has become a focus of concern in Canada. The recent identification of new variant CJD led to a review of the Canadian mortality database to identify any clustering of CJD by age, sex, or geographic location. The study by Holman and colleagues (1), which uses mortality data and active surveillance methods, has provided further information on the epidemiology of Creutzfeldt-Jacob disease (CJD). The study concluded that the incidence of the disease in the United States has remained stable and is similar to crude incidence worldwide at about one case per million annually. In addition, the study found no evidence that new variant CJD is present in the U.S. population. These findings are of interest because the epidemiology of CJD in Canada is not well described, and concerns have been raised there about the transmissibility of CJD through transfusion of blood and blood products or through tissue and organ transplants. In addition, many Canadians travel to and from the United Kingdom, where new variant CJD was first identified and linked to the bovine spongiform encephalopathy epidemic (2). We report here our findings on the epidemiology of CJD in Canada, which are derived from published mortality data (underlying cause of death by standard 5-year age group and sex, for all Canadian residents). The Statistics Canada mortality reports for the years 1979 to 1993 were reviewed for CJD deaths by sex and age group for each province and territory. Reports before 1979 were not used because CJD (ICD-9 code 46.1) was not listed as a cause of death before this time.

38. Section II : Groupe De Travail : Creutzfeldt-Jacob Division II Working Group on. creutzfeldtjacob disease. NAME, FIRSTNAME, ACTIVITY, ACTIVITY, E-MAIL. PASTORET P.Pierre, President, 04/366.42 http://www.health.fgov.be/CSH_HGR/English/Structure/Division_subdivision/divisio

Division II Working Group on Creutzfeldt-Jacob Disease NAME, FIRST NAME ACTIVITY ACTIVITY E-MAIL PASTORET P.Pierre President paul.pierre.pastoret@ulg.ac.be CONTENT Jean Member j.content@ben.ulb.ac.be CRAS Patrick Expert cras@uia.ua.ac.be CREVITS Luc Expert DE BLEECKER Jan Expert DE REUCK Jacques Expert DEPREZ Manuel Expert EBINGER Guy Expert FRANCK Georges Expert GODFRAIND Catherine Expert HILDEBRAND Jerzy Expert neurologe@ulb.ac.be MARTIN Jean-Jacques Expert jjmneuro@uia.ua.ac.be MICHOTTE Alex Expert RESNIK Michel Expert ROBBERECHT Wim Expert wim.robberecht@uz.kuleuven.ac.be SCIOT Raf Expert raf.sciot@uz.kuleuven.ac.be SINDIC Christian Expert sindic@mchm.ucl.be VAN DE VLOED Olga Expert VANDERHAEGHEM J.J. Expert DUBOIS Jean-Jacques Scientific Secretariat jjacques.dubois@health.fgov.be STROOBANT A. Invited Public of Officer andre.stroobant@ihe.be VAN LOOCK Walter Invited Public of Officer vanloock@intec.rug.ac.be

39. OLPA - Legislative Updates - Animal Disease Risk Assessment, Prevention, And Con the causes and mechanisms of transmission of foot and mouth disease and bovine spongiformencephalopathy (BSE), variant creutzfeldtjacob disease, and related http://olpa.od.nih.gov/legislation/107/publiclaws/planimaldisease.asp

FAQs Site Map Links Home April 6, 2003 Bill Tracking Legislative Updates Public Laws Hearings ... Committees of Interest to NIH OLPA 107th Congress Public Laws Other Legislation Animal Disease Risk Assessment, Prevention, and Control Act of 2001 P.L. 107-9 (S.700) Impact of Public Law Legislative History As part of his introductory remarks in the Congressional Record, Senator Ben Nighthorse Campbell (R-CO) noted that the purpose of S. 700 is to establish a Federal task force to prevent the spread of mad cow disease, foot and mouth disease, and related livestock diseases within the United States. On the same day Senator Campbell introduced S. 700, he testified at a hearing of the Senate Commerce Subcommittee on Consumer Affairs, Foreign Commerce and Tourism that addressed specific measures that should be taken in the United States to prevent BSE or mad cow disease. At the hearing, Senator Campbell said that Congress needed to do everything necessary to protect the livestock industry with careful contingency planning by creating a task force to report back to Congress on efforts and plans undertaken at agencies to prevent the spread of animal diseases and make recommendations for the future. On April 4, 2001, S. 700 was introduced by Senator Campbell as the Mad Cow and Related Diseases Prevention Act of 2001. On April 5, S. 700 was placed on the Senate Legislative Calendar for consideration. Senator Orrin Hatch (R-UT) offered an amendment in the nature of a substitute on the Senate floor, which was agreed to. The amendment changed the name of the bill to the Animal Disease Risk Assessment, Prevention, and Control Act of 2001 but did not substantively change the legislation. After passing the Senate under unanimous consent on April 5, 2001, the measure proceeded to the House for consideration. On May 9, 2001, the House passed S. 700 by voice vote and it was signed into law (P.L. 107-9) on May 24, 2001.

40. Creutzfeldt-Jacob Disease Encyclopedia Disease - C - creutzfeldt-jacob disease. Creutzfeldt-Jacobdisease. Causes and Risks Creutzfeldt-Jakob disease http://www.rwjuhh.net/Atoz/encyclopedia/article/000788.asp

For a complete list of hospital classes and events, click here to connect to HealthConnection Online Medical Encyclopedia Encyclopedia Disease C -> Creutzfeldt-Jacob disease Creutzfeldt-Jacob disease Causes and Risks: Creutzfeldt-Jakob disease is an organic brain syndrome caused by a viral-type organism. The disorder is rare, occurring in about 1 out of 1,000,000 people. It usually first appears in midlife, beginning between ages 20 and 68, with the average age at onset of symptoms being between 57 and 62. Cases have occurred in adolescents who have received growth hormone derived from cadavers, despite the production process, which is intended to kill all bacterial and viral material in the product. The causative agent, or prion, is thought to have been transmitted in the growth hormone (extracted from the pituitary of cadavers), which explains the abnormally early appearance of the disease. Cadaver-derived growth hormone has been replaced by synthetically manufactured growth hormone and contagion is no longer a problem. Once symptoms appear there is rapid progression of the disorder with progressive loss of brain function. The lesion and deterioration of function is similar to that of

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