Central Pontine Myelinolysis central pontine myelinolysis. A Hospital. A resource with informationon over 4000 medical topics including central pontine myelinolysis. http://www.marylandalzheimers.com/medical-terms/02060.htm
Irish Medical Journal A clinical diagnosis of central pontine myelinolysis (CPM) was made by the neurologists2 . On days 5 to 6 the patient developed diabetes insipidus (DI), and a http://www.imj.ie/news_detail.php?nNewsId=2490&nVolId=96
¤E¤Q¦~«×¤º±Mµ§¸ÕÃD - ¯«¸g¬ì D. Alcoholic cerebellar degeneration. E. central pontine myelinolysis. A. (encephalopathy). B. ?(central pontine myelinolysis). http://www.sim.org.tw/exam/exam90/q11.htm
uAavSTªV 477, central pontine myelinolysis1?. A CaseReport of central pontine myelinolysis Complicated by Diabetes477. http://www.jds.or.jp/mokuji/45/45_07.html
Annals Of Internal Medicine: Letters sodium levels (10 mmol/L in 12 hours) to twice the maximal rate recommended as optimalto avoid osmotic demyelination or central pontine myelinolysis (9 mmol/L http://www.annals.org/issues/v133n12/full/200012190-00023.html
Extractions: Ayus and colleagues' report of hyponatremic encephalopathy with noncardiogenic pulmonary edema in marathon runners demonstrates the life-threatening potential of this syndrome and its treatment. The authors' use of intravenous hypertonic sodium chloride (514 mmol/L) resulted in an increase in plasma sodium levels (10 mmol/L in 12 hours) to twice the maximal rate recommended as optimal to avoid osmotic demyelination or central pontine myelinolysis (9 mmol/L per 24 hours) . Was there evidence of this lesion at autopsy in the runner with the lowest sodium level (117 mmol/L) who developed brain stem herniation during treatment? Exercise-related hypovolemic hyponatremia, as shown by low pulmonary wedge pressures in this study, might be more appropriately treated with intravenous isotonic sodium chloride (308 mmol/L) to enhance extracellular fluid volume while more gradually correcting plasma tonicity in the high-risk setting of rapid-onset osmotic dysequilibrium . Better understanding of this rare syndrome and potential complications during treatment may lead to safer outcomes and preventive strategies. Arthur J. Siegel, MD
Myelinolysis, Central, Pontine myelinolysis, central, pontine,, Print this article, see central pontinemyelinolysis GS The Encyclopaedia of Medical Imaging Volume VI1, http://www.amershamhealth.com/medcyclopaedia/Volume VI 1/MYELINOLYSIS CENTRAL PO
ACR Learning File Web There is no mass effect noted. Return to top. Diagnosis central pontinemyelinolysis (CPM) (osmotic myelinolysis). Return to top. Discussion http://www.learningfile.com/learning_file/viewcase.php?section=nu&case_num=613
Extractions: Diseases Chapters: A Collection of High Quality Online Resources for Health Professionals Pathology see Pathology of Infectious Diseases and Disease of Myelin Overview Multiple Sclerosis Overview Multiple Sclerosis - Neurology Associates of Arlington Multiple Sclerosis - Praxis MD Multiple Sclerosis: Current Status and Strategies for the Future - National Academy Press, Multiple Sclerosis: Lectures - University of Utah, Houston (
Hyponatremic Encephalopathy - Medstudents - Nephrology The latter case is best illustrated by the entity called centralpontine myelinolysis or osmotic demyelination syndrome. http://www.medstudents.com.br/nefro/nefro1.htm
Extractions: Medstudents' Homepage Among all the electrolyte disorders commonly found in general medical practice, hyponatremia appears to be the most common abnormality. Its importance is due to its association with a vast array of signs and symptoms, of which, undobtedly, the neurologic manifestations are the ones that present the highest morbidity and mortality. Hyponatremia may be observed in a wide spectrum of clinical settings, the most common being: postoperative hyponatremia, renal failure, liver failure, heart failure, nephrotic syndrome and the syndrome of inappropriate secretion of vasopressin (SIADH). In the latter syndrome, the causes may be highly variable, ranging from the inappropriate secretion secondary to structural lesions ( tumors, CNS diseases, lung diseases ) to the one caused by certain drugs (antineoplastic, oral hypoglycemics, diuretics, clofibrate, carbamazepine and morphine-like analgesics). Other causes of the SIADH are chronic illnesses and the aquired immune deficiency syndrome, which courses frequently with hyponatremia. Neurologic manifestations Generally observed with plasma sodium levels lower than 130mmol/L, the neurologic manifestations of hyponatremia range from mild symptoms, such as headache, nausea and emesis to those more severe like seizures, coma and respiratory arrest. Psychiatric signs may be observed, the most common of which being a bizarre behavior of recent onset. Other manifestations tend to occur less frequently, but seem to be very characteristic of this entity called hyponatremic encephalopathy. These less common ocurrences are bradycardia, hypertension, hypothermia and dilated pupils. Urinary incontinence may occur, although much less frequently. Other signs and symptoms may be observed in protracted hyponatremia such as : weakness (focal or generalized), ataxia, asterixis, Babinski sign and delirium. Psychiatric manifestations in these cases may include depression, cognitive impairment and psychosis.
Katalog - Wirtualna Polska Serwis Katalog w Wirtualna Polska S.A. pierwszy portal w Polsce. http://katalog.wp.pl/DMOZ/Health/Conditions_and_Diseases/Neurological_Disorders/
úåãwG@2000N5E59ª5 The summary for this Japanese page contains characters that cannot be correctly displayed in this language/character set. http://www.med.nihon-u.ac.jp/department/NUMA/No59-5/MRI.html
